Fetal Bone Formation Is Decreased from Middle Pregnancy to Birth

Fetal Bone Formation Is Decreased from Middle Pregnancy to Birth.

Fetal bone development is a complex process that is regulated and maintained by minerals, hormones, and growth factors delivered from the mother via the placenta. Various biochemical markers of fetal bone development have been identified. However, many aspects of this process remain unclear. The aim of the study was to measure the activities of serum tartrate-resistant acid phosphatase type 5b ...

Relationship of mothers' fasting during pregnancy and fetal birth weight

Introduction: Fasting is a commitment to God, therefore many pregnant women have a tendency to perform it, but they are concerns about its possible complications which may affect fetal health. Objective: This study aimed to determine the relationship between fasting on pregnant women and their fetal birth weight. Method: This study was conducted on pregnant women referring to maternity hospital...

Human Fetal Brain Connectome: Structural Network Development from Middle Fetal Stage to Birth

Complicated molecular and cellular processes take place in a spatiotemporally heterogeneous and precisely regulated pattern in the human fetal brain, yielding not only dramatic morphological and microstructural changes, but also macroscale connectomic transitions. As the underlying substrate of the fetal brain structural network, both dynamic neuronal migration pathways and rapid developing fet...

The Value of Reporting Decreased or Absent Fetal Movements by Mothers in Predicting the Pregnancy Outcome

Parathyroid hormone is essential for normal fetal bone formation.

Parathyroid hormone (PTH) is a potent pharmacologic inducer of new bone formation, but no physiologic anabolic effect of PTH on adult bone has been described. We investigated the role of PTH in fetal skeletal development by comparing newborn mice lacking either PTH, PTH-related peptide (PTHrP), or both peptides. PTH-deficient mice were dysmorphic but viable, whereas mice lacking PTHrP died at b...